:fm: What corrupt individuals that are part of the FDA. Power hungry greedy individuals that need to be brought to justice!!! I can't wait til this system of things collapses all around them. What sweet justice it will be then!
:fm: What corrupt individuals that are part of the FDA. Power hungry greedy individuals that need to be brought to justice!!! I can't wait til this system of things collapses all around them. What sweet justice it will be then!
Before you all get too cranked up over this report, remember that you're hearing only the facts the story tellers which to be heard.
Consider for example: Stanislaw Burzynski and "Antineoplastons"
And for what it's worth I know more than one M.D. who has died of cancer. Another (my ex wife) who went through entirely normal Chemotherapy. And so on and so on.
25% (antineoplastons) vs. 9% (chemo/rad) in brain tumors...I wouldn't call it a cure, but I'd say it might be a step in the right direction.
As for the other half of the discussion...I'd have to say that IMHO Politics, corruption, and capitalism will always trump the "human factor" of any government decision.
25% (antineoplastons) vs. 9% (chemo/rad) in brain tumors...I wouldn't call it a cure, but I'd say it might be a step in the right direction.
I'd have to watch that part again, but I think it was .9% on the chemo/rad ... either way I would say your right .. not quite a cure, but definately better odds .. either way .. with the proper funding, who knows where this would have been today ...
Crowley
yes sir you are correct. hell we cant even "cure" the common cold, but we can treat the symptoms for a fair price. there is no money in the cure, only in the treatments.
This thread is full of low-hanging fruit.
If one company developed one singular prophylactic measure or cure for cancer, of any variety, they would market it as quickly as humanly possible and crush their competitors.
Oh wait...they did that. It's called Gardasil. Now there are conspiracy theorists that are convinced it's not a means of suppressing cancer, but a means of government injecting an unknown substance into your body under the guise of "helping people" only to control the masses like sheep.
Some people were born full-retard.
+1000
as a practicing physician, its downright scary the lack of intelligence in this thread. there's no FDA in other countries, why aren't these treatments widespread througout the rest of the world? i will agree that the FDA does significantly delay americans' access to medications and procedures while they (the FDA) make sure said medication/procedure is 'safe'.
is someone in this thread actually suggesting that less physicians die of cancer than the general public?? seriously??
spend a bit more time learning about the thousands of different cancers and how each one uniquely affects the body. you'll quickly realize that stating there's one cure for all types of cancer is simply insane. plenty of cancers are curable, plenty are not.
next time someone starts a thread stating "pray for my family member and the physicians trying to treat their cancer/heart attack/broken spine/stroke/etc", i'll post a link to this thread to see how many idiots want to talk shit
sure, i feel sorry for the police officer in the beginning of the video. unfortunately, he's being misinformed/mislead
Japanese scientists have tested antineoplastons A10 and AS2-1 in vitro for cell growth inhibition and progression in several human hepatocellular cell lines.[2,3] Tests were performed in a dose-dependent manner at concentrations varying from 0.5 to 8 µg/mL for A10 and AS2-1, and growth inhibition was generally observed at 6 to 8 µg/mL. This dose level is considered excessively high and generally reflects a lack of activity. Growth inhibition of one of the cell lines (KIM-1) was observed at low concentration for a mixture of cisplatin (CDDP) and A10, but this result was probably caused by the cisplatin, which was effective at concentrations of 0.5 to 2.0 µg/mL when tested alone.[4] AS2-1 was reported to induce apoptosis in three of the cell lines at concentrations of 2 and 4 µg/mL.
Antineoplaston A10 was also shown to inhibit prolactin or interleukin-2 stimulation of mitogenesis in a dose-dependent manner in rat Nb2 lymphoma cell line. The addition of A10 (1–12 mm) to prolactin-stimulated cells inhibited growth but was reversible when A10 was removed, suggesting a cytostatic rather than cytotoxic mechanism of action. A10 also showed no toxicity in a chromium release assay. DNA synthesis was also inhibited by A10.[5]
The ability of antineoplaston A3, isolated from urine and not an analog, to inhibit the growth of the HBL-100 human breast cancer cell line in vitro was investigated in a study that also examined the toxicity of A3 in Swiss white mice. Antineoplaston A3 inhibited colony formation in a dose-dependent manner over a dose range of 0.05, 0.1, 0.2, and 0.4 µg/mL.[6]
A somewhat different approach to the use of A10 was taken by researchers in Egypt. Taking the developer's initial ideas about the presence of A10 in the urine of patients, this study looked for the amount of A10 in the urine of 31 breastcancer patients and compared this to the amount in 17 healthy controls. They found significantly (P < .001) less A10 in the urine of breast cancer patients than in controls, suggesting that the amount of A10 in urine has a potential use as a screening tool.[7]
The same researchers looked at the immunomodulating potential of A10 by examining the inhibition of neutrophil apoptosis induced by A10 in vitro. Neutrophils from 28 breast cancer patients and 28 controls were obtained from blood samples. Urine samples were obtained from the same patients and tested for the presence of A10. Cancer patients had significantly (P < .001) higher levels of neutrophil apoptosis and significantly lower levels of A10. Neutrophil apoptosis was assessed by adding A10 at a dose of 10 µg/mL to the cellular suspensions of 42 breast cancer patients. Nontreated samples were used as controls. A10 was found to significantly inhibit neutrophil apoptosis (P < .0001).[8]
Several analogs of antineoplaston A10 have been synthesized and their antineoplastic activity tested against various cell lines. These include aniline mustard analogs of antineoplaston A10 and Mannich bases of antineoplaston A10.[9,10] These analogs showed improved in vitro antitumor activity over that of antineoplaston A10.